A cell that utilizes NHEJ to repair a double-stranded DNA break which occurred in the open reading frame of a gene is susceptible to indels resulting in a frame shift mutation and ultimately a non-functional protein. False True

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03-06-2021
Biology

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A cell that utilizes NHEJ to repair a double-stranded DNA break which occurred in the open reading frame of a gene is susceptible to indels resulting in a frame shift mutation and ultimately a non-functional protein. False True

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Luisvaschettoidn Luisvaschettoidn · Answer 1 · 2021-06-05T22:27:57-04:00

Answer:

True

Explanation:

The non-homologous end-joining (NHEJ) pathway is a DNA repair pathway used to repair double-strand breaks that can occur throughout all cell cycle phases. Conversely to the homologous recombination (HR) pathway, the NHEJ pathway does not need a homologous template at the DNA double-strand breaks ends and therefore NHEJ is an error-prone repair pathway that may cause mutations within the repaired region of the DNA. The NHEJ pathway is well-known to induce indels (i.e., insertions and deletions) mutations that shift the reading frame of the protein (i.e., frameshift mutations), thereby leading to a completely different translated protein from the original one.

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